human pancreatic cancer cells Search Results


human pancreatic cancer stem cells  (Celprogen Inc)
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Celprogen Inc human pancreatic cancer stem cells
Human Pancreatic Cancer Stem Cells, supplied by Celprogen Inc, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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human pancreatic cancer cell line complete media  (Celprogen Inc)
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Celprogen Inc human pancreatic cancer cell line complete media
Human Pancreatic Cancer Cell Line Complete Media, supplied by Celprogen Inc, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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pancreatic cancer stem cell extracellular matrix  (Celprogen Inc)
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Celprogen Inc pancreatic cancer stem cell extracellular matrix
Pancreatic Cancer Stem Cell Extracellular Matrix, supplied by Celprogen Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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pancreatic cancer cells  (Celprogen Inc)
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Celprogen Inc pancreatic cancer cells
Pancreatic Cancer Cells, supplied by Celprogen Inc, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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pancreatic cancer stem cell complete growth media  (Celprogen Inc)
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Celprogen Inc pancreatic cancer stem cell complete growth media
Fig. 5. Cellular uptake studies in monolayer and microtumor cultures of prostate and <t>pancreatic</t> cancer stem cells. Panels (A–F): prostate cancer stem cells (scale bar: 25 m), and
Pancreatic Cancer Stem Cell Complete Growth Media, supplied by Celprogen Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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xenograft pancreatic cancer stem cells cscs  (Celprogen Inc)
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Celprogen Inc xenograft pancreatic cancer stem cells cscs
Fig. 6 Interaction of the block copolymers and HDAC8-responsive nanoparticles with different types of PDAC cells and non-cancerous HPNE cells in vitro. (A) PEG-block-poly(acetylated L-lysine) block copolymers studied in this work do not trigger cytotoxicity in different cell lines. (B) Chemical structure of the STAT3 inhibitor, Napabucasin (NAPA), which has been used as the model hydrophobic drug to demonstrate the encapsulation and HDAC-mediated release activity of the nanoparticles in the context of drug delivery. (C) HDAC 8 (1 mM) triggers the release of NAPA, and over 90% of the encapsulated drug is released from these nanoparticles after incubation with the enzyme for 3 h. The standard deviation of mean is taken for N = 3 replicates. Without the enzyme, the drug release rate and extent were significantly decreased. (D) NAPA-loaded nanoparticles showed a concentration- dependent effect on different types of cancer cells, with a more prominent effect on cancer stem cells <t>(CSCs).</t>
Xenograft Pancreatic Cancer Stem Cells Cscs, supplied by Celprogen Inc, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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human pancreatic cancer cell lines panc1  (Servicebio Inc)
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Servicebio Inc human pancreatic cancer cell lines panc1
Fig. 6 Interaction of the block copolymers and HDAC8-responsive nanoparticles with different types of PDAC cells and non-cancerous HPNE cells in vitro. (A) PEG-block-poly(acetylated L-lysine) block copolymers studied in this work do not trigger cytotoxicity in different cell lines. (B) Chemical structure of the STAT3 inhibitor, Napabucasin (NAPA), which has been used as the model hydrophobic drug to demonstrate the encapsulation and HDAC-mediated release activity of the nanoparticles in the context of drug delivery. (C) HDAC 8 (1 mM) triggers the release of NAPA, and over 90% of the encapsulated drug is released from these nanoparticles after incubation with the enzyme for 3 h. The standard deviation of mean is taken for N = 3 replicates. Without the enzyme, the drug release rate and extent were significantly decreased. (D) NAPA-loaded nanoparticles showed a concentration- dependent effect on different types of cancer cells, with a more prominent effect on cancer stem cells <t>(CSCs).</t>
Human Pancreatic Cancer Cell Lines Panc1, supplied by Servicebio Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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panc-1 cells  (Korean Cell Line Bank)
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Korean Cell Line Bank panc-1 cells
Fig. 6 Interaction of the block copolymers and HDAC8-responsive nanoparticles with different types of PDAC cells and non-cancerous HPNE cells in vitro. (A) PEG-block-poly(acetylated L-lysine) block copolymers studied in this work do not trigger cytotoxicity in different cell lines. (B) Chemical structure of the STAT3 inhibitor, Napabucasin (NAPA), which has been used as the model hydrophobic drug to demonstrate the encapsulation and HDAC-mediated release activity of the nanoparticles in the context of drug delivery. (C) HDAC 8 (1 mM) triggers the release of NAPA, and over 90% of the encapsulated drug is released from these nanoparticles after incubation with the enzyme for 3 h. The standard deviation of mean is taken for N = 3 replicates. Without the enzyme, the drug release rate and extent were significantly decreased. (D) NAPA-loaded nanoparticles showed a concentration- dependent effect on different types of cancer cells, with a more prominent effect on cancer stem cells <t>(CSCs).</t>
Panc 1 Cells, supplied by Korean Cell Line Bank, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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pancreatic cancer cell line panc-1  (Procell Inc)
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Procell Inc pancreatic cancer cell line panc-1
Fig. 6 Interaction of the block copolymers and HDAC8-responsive nanoparticles with different types of PDAC cells and non-cancerous HPNE cells in vitro. (A) PEG-block-poly(acetylated L-lysine) block copolymers studied in this work do not trigger cytotoxicity in different cell lines. (B) Chemical structure of the STAT3 inhibitor, Napabucasin (NAPA), which has been used as the model hydrophobic drug to demonstrate the encapsulation and HDAC-mediated release activity of the nanoparticles in the context of drug delivery. (C) HDAC 8 (1 mM) triggers the release of NAPA, and over 90% of the encapsulated drug is released from these nanoparticles after incubation with the enzyme for 3 h. The standard deviation of mean is taken for N = 3 replicates. Without the enzyme, the drug release rate and extent were significantly decreased. (D) NAPA-loaded nanoparticles showed a concentration- dependent effect on different types of cancer cells, with a more prominent effect on cancer stem cells <t>(CSCs).</t>
Pancreatic Cancer Cell Line Panc 1, supplied by Procell Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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cell line mia paca-2  (BioResource International Inc)
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BioResource International Inc cell line mia paca-2
Fig. 6 Interaction of the block copolymers and HDAC8-responsive nanoparticles with different types of PDAC cells and non-cancerous HPNE cells in vitro. (A) PEG-block-poly(acetylated L-lysine) block copolymers studied in this work do not trigger cytotoxicity in different cell lines. (B) Chemical structure of the STAT3 inhibitor, Napabucasin (NAPA), which has been used as the model hydrophobic drug to demonstrate the encapsulation and HDAC-mediated release activity of the nanoparticles in the context of drug delivery. (C) HDAC 8 (1 mM) triggers the release of NAPA, and over 90% of the encapsulated drug is released from these nanoparticles after incubation with the enzyme for 3 h. The standard deviation of mean is taken for N = 3 replicates. Without the enzyme, the drug release rate and extent were significantly decreased. (D) NAPA-loaded nanoparticles showed a concentration- dependent effect on different types of cancer cells, with a more prominent effect on cancer stem cells <t>(CSCs).</t>
Cell Line Mia Paca 2, supplied by BioResource International Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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bxpc-3 human pancreatic cancer cell line  (Korean Cell Line Bank)
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Korean Cell Line Bank bxpc-3 human pancreatic cancer cell line
Fig. 6 Interaction of the block copolymers and HDAC8-responsive nanoparticles with different types of PDAC cells and non-cancerous HPNE cells in vitro. (A) PEG-block-poly(acetylated L-lysine) block copolymers studied in this work do not trigger cytotoxicity in different cell lines. (B) Chemical structure of the STAT3 inhibitor, Napabucasin (NAPA), which has been used as the model hydrophobic drug to demonstrate the encapsulation and HDAC-mediated release activity of the nanoparticles in the context of drug delivery. (C) HDAC 8 (1 mM) triggers the release of NAPA, and over 90% of the encapsulated drug is released from these nanoparticles after incubation with the enzyme for 3 h. The standard deviation of mean is taken for N = 3 replicates. Without the enzyme, the drug release rate and extent were significantly decreased. (D) NAPA-loaded nanoparticles showed a concentration- dependent effect on different types of cancer cells, with a more prominent effect on cancer stem cells <t>(CSCs).</t>
Bxpc 3 Human Pancreatic Cancer Cell Line, supplied by Korean Cell Line Bank, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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human pancreatic cancer cell line capan-1  (FUJIFILM)
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FUJIFILM human pancreatic cancer cell line capan-1
Fig. 6 Interaction of the block copolymers and HDAC8-responsive nanoparticles with different types of PDAC cells and non-cancerous HPNE cells in vitro. (A) PEG-block-poly(acetylated L-lysine) block copolymers studied in this work do not trigger cytotoxicity in different cell lines. (B) Chemical structure of the STAT3 inhibitor, Napabucasin (NAPA), which has been used as the model hydrophobic drug to demonstrate the encapsulation and HDAC-mediated release activity of the nanoparticles in the context of drug delivery. (C) HDAC 8 (1 mM) triggers the release of NAPA, and over 90% of the encapsulated drug is released from these nanoparticles after incubation with the enzyme for 3 h. The standard deviation of mean is taken for N = 3 replicates. Without the enzyme, the drug release rate and extent were significantly decreased. (D) NAPA-loaded nanoparticles showed a concentration- dependent effect on different types of cancer cells, with a more prominent effect on cancer stem cells <t>(CSCs).</t>
Human Pancreatic Cancer Cell Line Capan 1, supplied by FUJIFILM, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Fig. 5. Cellular uptake studies in monolayer and microtumor cultures of prostate and pancreatic cancer stem cells. Panels (A–F): prostate cancer stem cells (scale bar: 25 m), and

Journal: Colloids and surfaces. B, Biointerfaces

Article Title: Peptide-targeted, stimuli-responsive polymersomes for delivering a cancer stemness inhibitor to cancer stem cell microtumors.

doi: 10.1016/j.colsurfb.2017.12.036

Figure Lengend Snippet: Fig. 5. Cellular uptake studies in monolayer and microtumor cultures of prostate and pancreatic cancer stem cells. Panels (A–F): prostate cancer stem cells (scale bar: 25 m), and

Article Snippet: Prostate cancer stem cells were maintained in human prostate cancer stem cell complete growth media with serum and antibiotics and pancreatic cancer stem cell were maintained in human pancreatic cancer stem cell complete growth media with serum and antibiotics from Celprogen.

Techniques:

Fig. 6. Expression of neuropilin-1 in prostate and pancreatic cancer stem cells as determined

Journal: Colloids and surfaces. B, Biointerfaces

Article Title: Peptide-targeted, stimuli-responsive polymersomes for delivering a cancer stemness inhibitor to cancer stem cell microtumors.

doi: 10.1016/j.colsurfb.2017.12.036

Figure Lengend Snippet: Fig. 6. Expression of neuropilin-1 in prostate and pancreatic cancer stem cells as determined

Article Snippet: Prostate cancer stem cells were maintained in human prostate cancer stem cell complete growth media with serum and antibiotics and pancreatic cancer stem cell were maintained in human pancreatic cancer stem cell complete growth media with serum and antibiotics from Celprogen.

Techniques: Expressing

Fig. 7. The viability of prostate and pancreatic cancer stem cells in monolayer and spheroid cultures. (A) Monolayer cultures of prostate cancer stem cells, (B) monolayer cultures

Journal: Colloids and surfaces. B, Biointerfaces

Article Title: Peptide-targeted, stimuli-responsive polymersomes for delivering a cancer stemness inhibitor to cancer stem cell microtumors.

doi: 10.1016/j.colsurfb.2017.12.036

Figure Lengend Snippet: Fig. 7. The viability of prostate and pancreatic cancer stem cells in monolayer and spheroid cultures. (A) Monolayer cultures of prostate cancer stem cells, (B) monolayer cultures

Article Snippet: Prostate cancer stem cells were maintained in human prostate cancer stem cell complete growth media with serum and antibiotics and pancreatic cancer stem cell were maintained in human pancreatic cancer stem cell complete growth media with serum and antibiotics from Celprogen.

Techniques:

Fig. 9. Flow cytometry analysis of the effect of napabucasin on prostate (A) and pancreatic cancer stem cells (B) with Annexin V and PI staining.

Journal: Colloids and surfaces. B, Biointerfaces

Article Title: Peptide-targeted, stimuli-responsive polymersomes for delivering a cancer stemness inhibitor to cancer stem cell microtumors.

doi: 10.1016/j.colsurfb.2017.12.036

Figure Lengend Snippet: Fig. 9. Flow cytometry analysis of the effect of napabucasin on prostate (A) and pancreatic cancer stem cells (B) with Annexin V and PI staining.

Article Snippet: Prostate cancer stem cells were maintained in human prostate cancer stem cell complete growth media with serum and antibiotics and pancreatic cancer stem cell were maintained in human pancreatic cancer stem cell complete growth media with serum and antibiotics from Celprogen.

Techniques: Flow Cytometry, Staining

Fig. 6 Interaction of the block copolymers and HDAC8-responsive nanoparticles with different types of PDAC cells and non-cancerous HPNE cells in vitro. (A) PEG-block-poly(acetylated L-lysine) block copolymers studied in this work do not trigger cytotoxicity in different cell lines. (B) Chemical structure of the STAT3 inhibitor, Napabucasin (NAPA), which has been used as the model hydrophobic drug to demonstrate the encapsulation and HDAC-mediated release activity of the nanoparticles in the context of drug delivery. (C) HDAC 8 (1 mM) triggers the release of NAPA, and over 90% of the encapsulated drug is released from these nanoparticles after incubation with the enzyme for 3 h. The standard deviation of mean is taken for N = 3 replicates. Without the enzyme, the drug release rate and extent were significantly decreased. (D) NAPA-loaded nanoparticles showed a concentration- dependent effect on different types of cancer cells, with a more prominent effect on cancer stem cells (CSCs).

Journal: Journal of materials chemistry. B

Article Title: Design and evaluation of nanoscale materials with programmed responsivity towards epigenetic enzymes.

doi: 10.1039/d4tb00514g

Figure Lengend Snippet: Fig. 6 Interaction of the block copolymers and HDAC8-responsive nanoparticles with different types of PDAC cells and non-cancerous HPNE cells in vitro. (A) PEG-block-poly(acetylated L-lysine) block copolymers studied in this work do not trigger cytotoxicity in different cell lines. (B) Chemical structure of the STAT3 inhibitor, Napabucasin (NAPA), which has been used as the model hydrophobic drug to demonstrate the encapsulation and HDAC-mediated release activity of the nanoparticles in the context of drug delivery. (C) HDAC 8 (1 mM) triggers the release of NAPA, and over 90% of the encapsulated drug is released from these nanoparticles after incubation with the enzyme for 3 h. The standard deviation of mean is taken for N = 3 replicates. Without the enzyme, the drug release rate and extent were significantly decreased. (D) NAPA-loaded nanoparticles showed a concentration- dependent effect on different types of cancer cells, with a more prominent effect on cancer stem cells (CSCs).

Article Snippet: The fourth cell variant is patient-derived xenograft pancreatic cancer stem cells (CSCs) obtained from Celprogen.

Techniques: Blocking Assay, In Vitro, Encapsulation, Activity Assay, Incubation, Standard Deviation, Concentration Assay